CH 6 LECTURE EXAM REFERENCE SHEET
34 flashcards · Other · by Fatdoink1
ends in -blasts
produces more cells
ends in -cytes
maintains cells, doesn't produce them anymore
exogenous cartilage growth
In appositional (exogenous) growth, chondroblasts in the perichondrium add new cartilage to the outside edge of the existing cartilage. happens in childhood/adolescence
endogenous cartilage growth
In interstitial (endogenous) growth, chondrocytes in the center of the tissue divide and add more matrix in between the existing cells happens in the rest of adulthood
compact bone structure, function, and location
structure: Hard, bony matrix predominates; many osteocytes are located within lacunae that are distributed in a circular fashion around the central canals; small passageways connect adjacent lacunae Function: Provides great strength and support; forms a solid outer shell on bones that keeps them from being easily broken or punctured Location: Outer portions of all bones, the shafts of long bones
cancellous bone structure, function, and location
structure: SPONGY BONE Latticelike network of scaffolding characterized by trabeculae with large spaces between them filled with hemopoietic tissue; the osteocytes, or bone cells, are located within lacunae in the trabeculae Function: Acts as scaffolding to provide strength and support without the greater weight of compact bone Location: In the interior of the bones of the skull, vertebrae, sternum, and pelvis; in the ends of the long bones
types of bone ossification
intramembranous endochondral
endochondral ossification
FIRST cartilage is formed, then turned into bone
intramembranous ossification
bone is formed
calcitonin and PHT
antagonistic hormones that regulate the balance of calcium between bone and blood
what is bone remodeling
the ongoing replacement of bone tissue with new bone tissue bone is constantly replaced by osteoCLASTS and made new by osteoBLASTS
open (compound) bone fracture
fracture where there's an open wound on the skin near the bone
closed (simple) bone fracture
bone breaks but there's no open wound in the skin
incomplete bone fracture
fracture where bone is cracked or partially broken
complete bone fracture
fracture where the bone is in two separate pieces
spontaneous bone fracture
fractures that occur without precipitating factors and without the degree of trauma that is usually required to break a bone
comminuted fracture
bone breaks into MORE than two pieces
impacted fracture
a break where the ends are driven INTO each other
linear fracture
run parallel to the long axis of the bone (literally a line in the middle)
transverse fracture
perpendicular to the shaft (long part) of bone
spiral fracture
bone is twisted broken, has a spiral breakage around the bone
oblique fracture
bone is broken at an angle
dentate fracture
have rough, toothed and broken ends
stellate fracture
breakage lines radiate from a single point
greenstick fracture
fractures like a twig, most common in children who have flexible bones
hairline fracture
2 broken sections of bone DONT separate (ex: skull fractures)
rickets cause, symptoms
caused by vitamin D deficiency (which means no calcium)
scurvy cause, symptoms
caused by vitamin C deficiency (which means no collagen)
hematoma
a localized mass of blood released from blood vessels but confined within an organ or a space. (When a bone is fractured, the blood vessels in the bone and surrounding periosteum are damaged and a hematoma forms)
callus
a mass of bone tissue that forms at a fracture site (in attempt to connect the broken ends of bones)
callus ossification
callus is replaced by woven spongy bone through endochondral ossification. osteoblasts enter the internal callus and begin to produce bone. Eventually, the fibers and cartilage of the internal callus are replaced by woven spongy bone, which further stabilizes the broken bone.
effects of aging on bone loss
the bone matrix in an older bone is more brittle than in a younger bone because decreased collagen production results in relatively more mineral and less collagen fibers. With aging, the amount of matrix also decreases because the rate of matrix formation by osteoblasts becomes slower than the rate of matrix breakdown by osteoclasts. Significant loss of bone increases the likelihood of bone fractures.
effect of exercise on bones
bone becomes stronger
factors that effect bone growth
nutrition (vitamin C and D) genetics hormones (testostetrone, estrogen)